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Höf

Divers Tabac

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Whey Native AM Nutrition


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Merci à vous 3.

Du coup est-ce que c'est lié à la nicotine ou plus largement au tabac ? Autrement dit, est-ce que sous substitut nicotinique par exemple on encourt les mêmes risques ?

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Posted (edited)

Ca n'aide pas trop...

"Previously, we showed that nicotine could suppress anti-cancer activities of retinoids in lung cancer cells. We also found that the suppressive effect of nicotine is likely due to induction of nur77 by nicotine. In this study, we analyzed whether nur77 could regulate expression of retinoic acid receptor-beta (RAR), which is known to mediate the anti-cancer effect of retinoids.

Does nicotine inhibit anticancer activities of Vitamin A?

Attention,site fortement biaisé.

Etude avec reward ph34r.png

Edited by Höf

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j'aime bien ça

What these studies really point out is the folly of confusing an isolated element from a whole food with health benefits.

ph34r.png

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Mon dieu, un lecteur ! ph34r.png

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La substantifique moelle. ^^

Ca m'a tout l'air d'être des ayatollahs de la santé/hygiénistes notoires en même temps... ph34r.png

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you're scared, confess

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you're scared, confess

ange003.gif

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Guest kenpachi
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Mon dieu, un lecteur ! ph34r.png

c'est pas parce qu'on réponds pas qu'on ne lit pas ! D:

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Cela veut qu'il va falloir que fasse attention à ne pas poster de sources biaisées ph34r.png

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En rapport avec la cigarette :

"

Time to First Cigarette and 4-(Methylnitrosamino)-1-(3-Pyridyl)-1-Butanol (NNAL) Levels in Adult Smokers; National Health and Nutrition Examination Survey (NHANES), 2007–2010

Abstract

Background: The time to first cigarette (TTFC) is a good indicator of several dimensions of nicotine dependence. An early TTFC is also associated with increased lung and oral cancer risk. Our objective was to determine the relationship between TTFC and exposure to tobacco smoke carcinogens.

Methods: We conducted a cross-sectional analysis of a nationally representative subsample of smoking adults that had urinary samples analyzed for tobacco biomarkers. The study included 1,945 participants from the 2007–2008 and 2009–2010 National Health and Nutrition and Examination Survey. The main outcome measure was creatinine-adjusted urinary 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanol (NNAL) levels.

Results: The cigarette-per-day adjusted levels of NNAL were twice as high in participants who smoked within 5 minutes after waking than in participants who refrained from smoking for at least 1 hour (0.58 vs. 0.28 ng/mL, P < 0.001). In multivariate linear models, a shorter TTFC was significantly associated with increasing NNAL levels, after adjusting for cigarettes smoked per day (or cotinine), secondhand smoke exposure, age, sex, race/ethnicity, and other potential confounders.

Conclusions: These data show that in a nationally representative sample, there is a dose-dependent relationship between earlier smoking in the day and higher biologic exposure to a tobacco smoke carcinogen.

Impact: Our study provides further evidence that highlights the relationship between TTFC, nicotine dependence, and cancer risk. Cancer Epidemiol Biomarkers Prev; 22(4); 1–8. ©2013 AACR."

Des scientifiques de l'Université d'État de Pennsylvanie ont constaté que les fumeurs qui ont commencé à fumer dans les 5 minutes après leurs réveil avait 2x plus grandes quantités de la 4 - (cancérogène méthylnitrosamino) -1 - (3-pyridyl)-1-butanol (NNAL) dans leur sang que les participants qui se sont abstenus de fumer pendant au moins 1 heure.

= Une personne qui fume sa cigarette après le réveil aura un risque accru d'acquérir cancer du poumon ou orale plutôt que d'attendre une heure pour s'en griller une.Et ce,indépendamment du nombre de cigarettes fumées par jour.

Edited by Höf

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Guest mamouth
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= Une personne qui fume sa cigarette après le réveil aura un risque accru d'acquérir cancer du poumon ou orale plutôt que d'attendre une heure pour s'en griller une.Et ce,indépendamment du nombre de cigarettes fumées par jour.

Conclusion : Le mieux c'est encore de ne pas fumer du toutbiggrin.png .

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News.

Effets sur la thyroïde.

"Clin Endocrinol (Oxf). 2013 Apr 13. doi: 10.1111/cen.12222. [Epub ahead of print]
Smoking And Thyroid.
Source

Department of Endocrinology & Metabolism, Academic Medical Center, University of Amsterdam, Netherlands.

Abstract

Current smoking in population surveys is associated with a slight dose-dependent fall of serum TSH, likely secondary to a rise of serum FT4 and FT3 induced by activation of the sympathetic nervous system; it is independent of iodine intake. In contrast, the slightly greater thyroid size in smokers is observed in iodine-deficient but not in iodine-sufficient areas, and caused by competitive inhibition of thyroidal iodide uptake by thiocyanate. Smokers have an increased prevalence of nontoxic goitre and thyroid multinodularity, at least in iodine deficient areas. Current smoking reduces dose-dependently the risk of thyroid cancer, which is more pronounced for papillary than for follicular types; the risk in former smokers approaches that of never smokers. The lower TSH and lower body mass index in smokers might contribute to this reduced risk. Current smoking lowers the risk of developing thyroid peroxidase and thyroglobulin antibodies, and subclinical and overt autoimmune hypothyroidism; the effect is dose-dependent, but disappears within 3 years after quitting smoking. There is evidence from an animal model of experimental autoimmune thyroiditis that anti-inflammatory effects of nicotine are involved. In contrast, smoking is a dose-dependent risk factor for Graves' hyperthyroidism and especially for Graves' ophthalmopathy. Smoking is related to a higher recurrence rate of Graves' hyperthyroidism, a higher risk on Graves' ophthalmopathy after 131I therapy, and a less favourable outcome of GO treatment with steroids or retrobulbar irradiation. The observed associations with smoking likely indicate causal relationships in view of consistent associations across studies, the presence of dose-response effects, and disappearance of associations after cessation of smoking. This article is protected by copyright. All rights reserved.

This article is protected by copyright. All rights reserved.

Fumer = pas bon,pas bon.

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Guest Oujda
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Comment la cigarette augmente le catabolisme musculaire ?

Identification of possible cigarette smoke constituents responsible for muscle catabolism
Oren Rom, Sharon Kaisari, Dror Aizenbudet Abraham Z. Reznick Journal of Muscle Research and Cell Motility volume 33, Numbers 3-4 (2012), 199-208

The age-related loss of muscle mass and strength also known as sarcopenia is significantly influenced by life style factors such as physical inactivity and impaired nutrition. Cigarette smoking is another life style habit that has been shown to be associated with sarcopenia and to affect skeletal muscle. Even today, smoking is still prevalent worldwide and is probably the most significant source of toxic chemicals exposure to humans. Cigarette smoke (CS) is a complex aerosol consisting of thousands of various constituents including reactive oxygen and nitrogen free radicals, toxic aldehydes and more. Previous epidemiological studies have identified tobacco smoking as a risk factor for sarcopenia. Clinical, in vivo and in vitro studies have revealed

CS-induced skeletal muscle damage due to impaired muscle metabolism, increased inflammation and oxidative stress, over-expression of atrophy related genes and activation of various intracellular signaling pathways.

This review aims to discuss and identify the components of CS that may promote catabolism of skeletal muscle.

Augmentation de l'inflammation et le stress oxydatif, la surexpression des gènes liés à l'atrophie et l'activation de diverses voies de signalisation intracellulaire.

Edited by Oujda

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Je crois l'avoir déjà vu chez MDG cette étude :)

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"Nicotine from edible Solanaceae and risk of Parkinson disease

Article first published online: 9 MAY 2013

DOI: 10.1002/ana.23884

Objective

To test whether risk of Parkinson disease (PD) is associated with consumption of nicotine-containing edibles from the same botanical family as tobacco, Solanaceae, including peppers, tomatoes, and potatoes.

Methods

In a population-based study with 490 newly diagnosed idiopathic PD cases diagnosed during 1992–2008 at the University of Washington Neurology Clinic or Group Health Cooperative in western Washington State and 644 unrelated, neurologically normal controls, we examined whether PD was associated with self-reported typical frequency of consumption of peppers, tomatoes, tomato juice, and potatoes during adulthood, while adjusting for consumption of other vegetables, age, sex, race/ethnicity, tobacco use, and caffeine.

Results

PD was inversely associated with consumption of all edible Solanaceae combined (relative risk [RR] = 0.81, 95% confidence interval [CI] = 0.65–1.01 per time per day), but not consumption of all other vegetables combined (RR = 1.00, 95% CI = 0.92–1.10). The trend strengthened when we weighted edible Solanaceae by nicotine concentration (ptrend = 0.004).

An inverse association was also evident for peppers specifically (ptrend = 0.005). The potentially protective effect of edible Solanaceae largely occurred in men and women who had never used tobacco or who had smoked cigarettes <10 years.

Interpretation

Dietary nicotine or other constituents of tobacco and peppers may reduce PD risk. However, confirmation and extension of these findings are needed to strengthen causal inferences that could suggest possible dietary or pharmaceutical interventions for PD prevention. Ann Neurol 2013"

La nicotine des poivrons ( et les poivrons en général) contre Parkison.

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Smoking ban in public areas is associated with a reduced incidence of hospital admissions due to ST-elevation myocardial infarctions in non-smokers. Results from the BREMEN STEMI REGISTRY

Abstract

Introduction

Laws banning tobacco smoking from public areas have been passed in several countries, including the region of Bremen, Germany at the end of 2007. The present study analyses the incidence of hospital admissions due to ST-elevation myocardial infarctions (STEMIs) before and after such a smoking ban was implemented, focusing on differences between smokers and non-smokers. In this respect, data of the Bremen STEMI Registry (BSR) give a complete epidemiological overview of a region in northwest Germany with approximately 800,000 inhabitants since all STEMIs are admitted to one central heart centre.

Methods and results

Between January 2006 and December 2010, data from the BSR was analysed focusing on date of admission, age, gender, and prior nicotine consumption. A total of 3545 patients with STEMI were admitted in the Bremen Heart Centre during this time period. Comparing 2006–2007 vs. 2008–2010, hence before and after the smoking ban, a 16% decrease of the number of STEMIs was observed: from a mean of 65 STEMI/month in 2006–2007 to 55/month in 2008–2010 (p < 0.01). The group of smokers showed a constant number of STEMIs: 25/month in 2006–2007 to 26/month in 2008–2010 (+4%, p = 0.8). However, in non-smokers, a significant reduction of STEMIs over time was found: 39/month in 2006–2007 to 29/month in 2008–2010 (−26%, p < 0.01). The decline of STEMIs in non-smokers was consistently observed in all age groups and both sexes. Adjusting for potentially confounding factors like hypertension, obesity, and diabetes mellitus did not explain the observed decline.

Conclusions

In the BSR, a significant decline of hospital admissions due to STEMIs in non-smokers was observed after the smoking ban in public areas came into force. No reduction of STEMI-related admissions was found in smokers. These results may be explained by the protection of non-smokers from passive smoking and the absence of such an effect in smokers by the dominant effect of active smoking.

L' Interdiction de fumer en public pourrait sauver des vies de non-fumeurs.

Les résultats allemands de l'interdiction publiques offre une réduction de 26% du nombre d'hospitalisations dues à l'élévation du segment ST/ infarctus du myocarde et ce n'est pas chez les fumeurs, mais chez les non-fumeurs qui ne sont désormais plus exposés au tabagisme dans les bars et restaurants.

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Guest plue
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ouais sérieux ça deviens grave des fois je suis dans un café tellement y'a des gens qui fume l'endroit se transforme en bain maure

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GABA et glutamate pour arrêter du fumer.

Assez complexe mais complet.

Full.

The “Stop” and “Go” of Nicotine Dependence: Role of GABA and Glutamate

Abstract

Nicotine plays an important role in the initiation and maintenance of tobacco smoking.

Importantly, chronic nicotine exposure alters the function of brain reward systems, resulting in the development of a nicotine-dependent state. This nicotine-dependent state is associated with aversive affective and somatic signs upon abstinence from smoking, often leading to relapse in abstinent smokers.

This article reviews the role of the major excitatory and inhibitory neurotransmitters glutamate and γ-aminobutyric acid (GABA), respectively, in both the reinforcing effects of nicotine and development of nicotine dependence. Evidence suggests that blockade of glutamatergic neurotransmission attenuates both nicotine intake and nicotine seeking. In contrast, both nicotine intake and nicotine seeking are attenuated when GABA neurotransmission is facilitated.

In conclusion, medications that either attenuate/negatively modulate glutamatergic neurotransmission or facilitate/positively modulate GABA neurotransmission may be useful for promoting smoking cessation in humans.

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Le markenting du tabac au top.


When a range of well known influences for taking up smoking was factored in, smoking among peers proved the strongest influence, followed closely by exposure to tobacco ads.

The greater the exposure to tobacco ads, the greater was the likelihood that the teen would take up smoking, the analysis showed.

Teens who saw the most tobacco ads (11 to 55) were around twice as likely to become established smokers and daily smokers as those who saw the least (0 to 2.5).

And for each additional 10 sightings of a tobacco ad, a teen was 38% more likely to become an established smoker, and 30% more likely to smoke every day compared with sightings for non-tobacco product ads.

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Parental smoking during pregnancy and risk of overweight and obesity in the daughter

Abstract

Objective:

Emerging evidence suggests that prenatal exposures may affect long-term health outcomes. In utero exposure to smoking is associated with an increased risk of overweight and obesity in children and adolescents. However, few studies have examined how prenatal exposure to parental smoking influences risk of obesity in adulthood and whether these associations are independent of childhood and adolescent adiposity. The aim of the current study was to investigate whether prenatal exposure to parental smoking influences body size in adulthood and whether any association may be mediated by childhood and adolescent body size.

Methods:

We investigated the association between parental smoking during pregnancy and risk of overweight and obesity in adulthood and at age 18, and adiposity during childhood among 35370 participants in the Nurses’ Health Study II. Data on smoking during pregnancy and socioeconomic variables were provided by the mothers, and anthropometric data and adult risk factors were reported by participants.

Results:

After adjustment for socioeconomic and behavioral variables, maternal smoking during pregnancy was associated with adiposity at ages 5-10, age 18, and during adulthood. For age 18 overweight the ORs (95%CIs) for 1–14, 15–24, and 25+cigarettes/day were 1.13 (1.18–1.50), 1.40 (1.20–1.64), and 1.15 (0.79–1.69) and for obesity were 1.41 (1.14–1.75), 1.69 (1.31–2.18), and 2.36 (1.44–3.86). The corresponding ORs (95% CIs) for obesity in adulthood were 1.26 (1.16–1.37), 1.46 (1.30–1.63), and 1.43 (1.10–1.86). Risk of adiposity was not increased among daughters whose mothers stopped smoking during the first trimester (OR [95% CI] for overweight (1.03 [95% CI 0.90–1.17] and obesity (1.12 [95% CI 0.97–1.30]). Women whose fathers smoked during pregnancy were also at increased risk of overweight and obesity in adulthood with covariate-adjusted ORs (95% CIs) for obesity of 1.19 (1.11–1.29) for 1–14 cigarettes/day, 1.27 (1.18–1.37) for 15–24 cigarettes/day, and 1.40 (1.27–1.54) for 25+ cigarettes/day compared to fathers who did not smoke (ptrend<0.0001). Paternal smoking during pregnancy was also associated with an increased risk of obesity at age 18 among those whose fathers smoked 15 or more cigarettes/day but was not associated with childhood body size.

Conclusions:

Maternal smoking during pregnancy was associated in a dose-response manner with overweight and obesity in the daughter through adolescence and adult life. Smoking cessation during the first trimester appears to mitigate this excess risk. Paternal smoking was also associated with risk of overweight and obesity of the adult daughter and this association persisted after adjustment for maternal smoking.

Petite news.

Les mères fumeuses ont des filles obèses:

Après ajustement pour les variables socio-économiques et comportementales, le tabagisme maternel pendant la grossesse a été associé à l'adiposité à 5-10 ans, 18 ans et l'âge adulte.

Pour 18 ans en surpoids / obésité

1-14 cigarettes / jour: +13% / +41%

15-25 cig / jour: +40% / +69%

25 + cig / jour: +15% / +136%

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Guest albator59
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je fumer plus de 60 clopes par jour

je ne prener quasiment pas de muscle

j ai arrêter mais j ai eut plusieurs tendinites qui mon fait arrêter la muscu pendant presque 10 ans

quand j ai repris avec les même exercice et le même régime

j ai pris en 7 mois ce que je n avez jamais réussie a prendre en 3 ans

il est vrai que le cas est extrême

mais on voie la diferance significative entre fumer et ne pas fumer

de plus je serais déjà mort du cancer ou autre depuis le temps

le 8 octobre cela fera 11 ans

et c est encore dure et a des moments plus que d autres

courage pour arrêter il faut le vouloir et avoir de la volonter

je recommande de trainer avec des fumeurs, on garde l odeur de tabac

on a l impression de prendre une bouffer même si on ne fume pas

mais bon moi cela était du temps ou l on pouver fumer partout

maintenant ce n est plus la même chose

je connais des gens qui on essailler de vapoter il on tousses repris

mais si on aissait pas on ne peut pas savoir

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Cigarette smoking and obesity are associated with decreased fat perception in women - Pepino - Obesity - Wiley Online Library

Conclusion: The ability to perceive fat and sweetness in and derive pleasure from foods is particularly compromised in obese women who smoke, which could contribute to excess calorie intake in this population already at high risk for cardiovascular and metabolic disease. Retronasal olfaction appears not to contribute to blunted flavor perception observed in obese smokers.

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Merci pour l'étude, ça ne m'étonne pas. Avec la "gueule pavée" faut rajouter du gras et sucre pour augmenter l'appétence (technique de la bouffe industrielle, fastfood etc).

Depuis que j'ai retrouvé le gout et l'odorat j'apprécie de nouveau beaucoup mieux tout ce qui légumes par exemple.

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Associations of physical activity and sport and exe... [Prev Med. 2014] - PubMed - NCBI

Les sportifs ne fument pas... Mais ils boivent.

En tout cas les suisses.

At baseline, logistic regression indicated that sport and exercise is negatively associated with at-risk use of cigarettes and cannabis. A positive association was obtained between physical activity and at-risk alcohol use. At baseline, sport and exercise was negatively associated with at-risk use of cigarettes and cannabis at follow-up. Adjusted for sport and exercise, physical activity was positively associated with at-risk use of cigarettes and cannabis.

CONCLUSION:

Sport and exercise is cross-sectionally and longitudinally associated with a low prevalence of at-risk use of cigarettes and cannabis. This protective effect was not observed for physical activity broadly defined. Taking a substance use prevention perspective, the promotion of sport and exercise among young adults should be encouraged.

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Une nouvelle méthode si vous essayez d'arrêter de fumer : Faire de nouvelles activités, "auto-expansibles" qui peuvent aider à soulager le besoin en nicotine. Des activités comme des puzzles ou des jeux avec un partenaire.

ISU Headlines » Idaho State’s Xu collaborates on study suggesting self-expanding activities can help with nicotine craving

PLOS ONE: An fMRI Study of Nicotine-Deprived Smokers' Reactivity to Smoking Cues during Novel/Exciting Activity

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Cigarette smoking has been shown to increase [...] lung volume

Faut de la place pour stocker 1 kilo de goudron.

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✅ Découvrez la boutique et les NOUVEAUX suppléments All-musculation ❗️

Compléments AM Nutrition ? Coaching H & F ? Le livre AM ? Accessoires ?️‍♀️



  • Boutique All-musculation


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    1. 200 pages de conseils entrainement, nutrition, programmes, compléments alimentaire !

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